kca3.1 channel (Wulff labs)
90
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Wulff labs
kca3.1 channel
Kca3.1 Channel, supplied by Wulff labs, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/kca3%2E1+channel/kca3+1+channel/pm36622407-182-0-21
Average 90 stars, based on 1 article reviews
Kca3.1 Channel, supplied by Wulff labs, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/kca3%2E1+channel/kca3+1+channel/pm36622407-182-0-21
Average 90 stars, based on 1 article reviews
kca3.1 channel - by Bioz Stars,
2026-09
90/100 stars
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Activation Assay:Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis. Article Snippet: Accepted Manuscript Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Govindaraj Anumanthan, Philip J. Wilson, Ratnakar Tripathi, Nathan P. Hesemann, Rajiv R. Mohan PII: S0014-4835(17)30534-1 DOI: 10.1016/j.exer.2017.12.003 Reference: YEXER 7243 To appear in: Experimental Eye Research Received Date: 26 July 2017 Accepted Date: 10 December 2017 Please cite this article as: Anumanthan, G., Wilson, P.J., Tripathi, R., Hesemann, N.P., Mohan, R.R., Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis, Experimental Eye Research (2018), doi: 10.1016/j.exer.2017.12.003.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Article Snippet: KCa3.1 channel is the major contributor to Ca 2+ influx in Article Title: KCa3.1 Mediates Dysregulation of Mitochondrial Quality Control in Diabetic Kidney Disease. Article Snippet: Hence, KCa3.1 has been proposed as a potential therapeutic target for sickle cell anemia, autoimmunity, and Article Title: Functional Potassium Channels in Macrophages. Article Snippet: Macrophages are the predominant component of innate immunity, which is an important protective barrier of our body.. Macrophages are present in all organs and tissues of the body, their main functions include immune surveillance, bacterial killing, tissue remodeling and repair, and clearance of cell debris.. In addition, macrophages can present antigens to T cells and facilitate inflammatory response by releasing cytokines. Article Title: Targeting Effector Memory T Cells with a Selective Peptide Inhibitor of Kv1.3 Channels for Therapy of Autoimmune Diseases Article Snippet: It is noteworthy that it was 1600-fold Article Title: Protein Kinase A-Mediated Suppression of the Slow Afterhyperpolarizing KCa3.1 Current in Temporal Lobe Epilepsy Article Snippet: The KCa3.1 channel is ubiquitously expressed in the plasma membrane of neural and Article Title: The Kv1.3 K + channel in the immune system and its "precision pharmacology" using peptide toxins. Article Snippet: The complex interplay of ion channel activities generates a characteristic Ca2+ signal in activated T-cells (right middle of the cell) that regulates a plethora of cellular processes, leading to cell activation and cell type-specific effector functions 1 3 in activated CCR7− effector memory T (TEM) cells, whereas CCR7+ naïve and central memory T (TCM) cells preferentially express the Transmission Electron Microscopy:Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis. Article Snippet: Accepted Manuscript Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Govindaraj Anumanthan, Philip J. Wilson, Ratnakar Tripathi, Nathan P. Hesemann, Rajiv R. Mohan PII: S0014-4835(17)30534-1 DOI: 10.1016/j.exer.2017.12.003 Reference: YEXER 7243 To appear in: Experimental Eye Research Received Date: 26 July 2017 Accepted Date: 10 December 2017 Please cite this article as: Anumanthan, G., Wilson, P.J., Tripathi, R., Hesemann, N.P., Mohan, R.R., Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis, Experimental Eye Research (2018), doi: 10.1016/j.exer.2017.12.003.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Article Snippet: KCa3.1 channel is the major contributor to Ca 2+ influx in Article Title: KCa3.1 Mediates Dysregulation of Mitochondrial Quality Control in Diabetic Kidney Disease. Article Snippet: Hence, KCa3.1 has been proposed as a potential therapeutic target for sickle cell anemia, autoimmunity, and Article Title: Functional Potassium Channels in Macrophages. Article Snippet: Macrophages are the predominant component of innate immunity, which is an important protective barrier of our body.. Macrophages are present in all organs and tissues of the body, their main functions include immune surveillance, bacterial killing, tissue remodeling and repair, and clearance of cell debris.. In addition, macrophages can present antigens to T cells and facilitate inflammatory response by releasing cytokines. Article Title: Targeting Effector Memory T Cells with a Selective Peptide Inhibitor of Kv1.3 Channels for Therapy of Autoimmune Diseases Article Snippet: It is noteworthy that it was 1600-fold Article Title: Protein Kinase A-Mediated Suppression of the Slow Afterhyperpolarizing KCa3.1 Current in Temporal Lobe Epilepsy Article Snippet: The KCa3.1 channel is ubiquitously expressed in the plasma membrane of neural and Article Title: The Kv1.3 K + channel in the immune system and its "precision pharmacology" using peptide toxins. Article Snippet: The complex interplay of ion channel activities generates a characteristic Ca2+ signal in activated T-cells (right middle of the cell) that regulates a plethora of cellular processes, leading to cell activation and cell type-specific effector functions 1 3 in activated CCR7− effector memory T (TEM) cells, whereas CCR7+ naïve and central memory T (TCM) cells preferentially express the Transfection:Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis. Article Snippet: Accepted Manuscript Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Govindaraj Anumanthan, Philip J. Wilson, Ratnakar Tripathi, Nathan P. Hesemann, Rajiv R. Mohan PII: S0014-4835(17)30534-1 DOI: 10.1016/j.exer.2017.12.003 Reference: YEXER 7243 To appear in: Experimental Eye Research Received Date: 26 July 2017 Accepted Date: 10 December 2017 Please cite this article as: Anumanthan, G., Wilson, P.J., Tripathi, R., Hesemann, N.P., Mohan, R.R., Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis, Experimental Eye Research (2018), doi: 10.1016/j.exer.2017.12.003.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Article Snippet: KCa3.1 channel is the major contributor to Ca 2+ influx in Article Title: KCa3.1 Mediates Dysregulation of Mitochondrial Quality Control in Diabetic Kidney Disease. Article Snippet: Hence, KCa3.1 has been proposed as a potential therapeutic target for sickle cell anemia, autoimmunity, and Article Title: Functional Potassium Channels in Macrophages. Article Snippet: Macrophages are the predominant component of innate immunity, which is an important protective barrier of our body.. Macrophages are present in all organs and tissues of the body, their main functions include immune surveillance, bacterial killing, tissue remodeling and repair, and clearance of cell debris.. In addition, macrophages can present antigens to T cells and facilitate inflammatory response by releasing cytokines. Article Title: Targeting Effector Memory T Cells with a Selective Peptide Inhibitor of Kv1.3 Channels for Therapy of Autoimmune Diseases Article Snippet: It is noteworthy that it was 1600-fold Article Title: Protein Kinase A-Mediated Suppression of the Slow Afterhyperpolarizing KCa3.1 Current in Temporal Lobe Epilepsy Article Snippet: The KCa3.1 channel is ubiquitously expressed in the plasma membrane of neural and Article Title: The Kv1.3 K + channel in the immune system and its "precision pharmacology" using peptide toxins. Article Snippet: The complex interplay of ion channel activities generates a characteristic Ca2+ signal in activated T-cells (right middle of the cell) that regulates a plethora of cellular processes, leading to cell activation and cell type-specific effector functions 1 3 in activated CCR7− effector memory T (TEM) cells, whereas CCR7+ naïve and central memory T (TCM) cells preferentially express the Incubation:Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis. Article Snippet: Accepted Manuscript Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Govindaraj Anumanthan, Philip J. Wilson, Ratnakar Tripathi, Nathan P. Hesemann, Rajiv R. Mohan PII: S0014-4835(17)30534-1 DOI: 10.1016/j.exer.2017.12.003 Reference: YEXER 7243 To appear in: Experimental Eye Research Received Date: 26 July 2017 Accepted Date: 10 December 2017 Please cite this article as: Anumanthan, G., Wilson, P.J., Tripathi, R., Hesemann, N.P., Mohan, R.R., Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis, Experimental Eye Research (2018), doi: 10.1016/j.exer.2017.12.003.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Article Snippet: KCa3.1 channel is the major contributor to Ca 2+ influx in Article Title: KCa3.1 Mediates Dysregulation of Mitochondrial Quality Control in Diabetic Kidney Disease. Article Snippet: Hence, KCa3.1 has been proposed as a potential therapeutic target for sickle cell anemia, autoimmunity, and Article Title: Functional Potassium Channels in Macrophages. Article Snippet: Macrophages are the predominant component of innate immunity, which is an important protective barrier of our body.. Macrophages are present in all organs and tissues of the body, their main functions include immune surveillance, bacterial killing, tissue remodeling and repair, and clearance of cell debris.. In addition, macrophages can present antigens to T cells and facilitate inflammatory response by releasing cytokines. Article Title: Targeting Effector Memory T Cells with a Selective Peptide Inhibitor of Kv1.3 Channels for Therapy of Autoimmune Diseases Article Snippet: It is noteworthy that it was 1600-fold Article Title: Protein Kinase A-Mediated Suppression of the Slow Afterhyperpolarizing KCa3.1 Current in Temporal Lobe Epilepsy Article Snippet: The KCa3.1 channel is ubiquitously expressed in the plasma membrane of neural and Article Title: The Kv1.3 K + channel in the immune system and its "precision pharmacology" using peptide toxins. Article Snippet: The complex interplay of ion channel activities generates a characteristic Ca2+ signal in activated T-cells (right middle of the cell) that regulates a plethora of cellular processes, leading to cell activation and cell type-specific effector functions 1 3 in activated CCR7− effector memory T (TEM) cells, whereas CCR7+ naïve and central memory T (TCM) cells preferentially express the Sequencing:Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis. Article Snippet: Accepted Manuscript Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Govindaraj Anumanthan, Philip J. Wilson, Ratnakar Tripathi, Nathan P. Hesemann, Rajiv R. Mohan PII: S0014-4835(17)30534-1 DOI: 10.1016/j.exer.2017.12.003 Reference: YEXER 7243 To appear in: Experimental Eye Research Received Date: 26 July 2017 Accepted Date: 10 December 2017 Please cite this article as: Anumanthan, G., Wilson, P.J., Tripathi, R., Hesemann, N.P., Mohan, R.R., Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis, Experimental Eye Research (2018), doi: 10.1016/j.exer.2017.12.003.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Article Snippet: KCa3.1 channel is the major contributor to Ca 2+ influx in Article Title: KCa3.1 Mediates Dysregulation of Mitochondrial Quality Control in Diabetic Kidney Disease. Article Snippet: Hence, KCa3.1 has been proposed as a potential therapeutic target for sickle cell anemia, autoimmunity, and Article Title: Functional Potassium Channels in Macrophages. Article Snippet: Macrophages are the predominant component of innate immunity, which is an important protective barrier of our body.. Macrophages are present in all organs and tissues of the body, their main functions include immune surveillance, bacterial killing, tissue remodeling and repair, and clearance of cell debris.. In addition, macrophages can present antigens to T cells and facilitate inflammatory response by releasing cytokines. Article Title: Targeting Effector Memory T Cells with a Selective Peptide Inhibitor of Kv1.3 Channels for Therapy of Autoimmune Diseases Article Snippet: It is noteworthy that it was 1600-fold Article Title: Protein Kinase A-Mediated Suppression of the Slow Afterhyperpolarizing KCa3.1 Current in Temporal Lobe Epilepsy Article Snippet: The KCa3.1 channel is ubiquitously expressed in the plasma membrane of neural and Article Title: The Kv1.3 K + channel in the immune system and its "precision pharmacology" using peptide toxins. Article Snippet: The complex interplay of ion channel activities generates a characteristic Ca2+ signal in activated T-cells (right middle of the cell) that regulates a plethora of cellular processes, leading to cell activation and cell type-specific effector functions 1 3 in activated CCR7− effector memory T (TEM) cells, whereas CCR7+ naïve and central memory T (TCM) cells preferentially express the Clinical Proteomics:Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis. Article Snippet: Accepted Manuscript Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Govindaraj Anumanthan, Philip J. Wilson, Ratnakar Tripathi, Nathan P. Hesemann, Rajiv R. Mohan PII: S0014-4835(17)30534-1 DOI: 10.1016/j.exer.2017.12.003 Reference: YEXER 7243 To appear in: Experimental Eye Research Received Date: 26 July 2017 Accepted Date: 10 December 2017 Please cite this article as: Anumanthan, G., Wilson, P.J., Tripathi, R., Hesemann, N.P., Mohan, R.R., Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis, Experimental Eye Research (2018), doi: 10.1016/j.exer.2017.12.003.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Article Snippet: KCa3.1 channel is the major contributor to Ca 2+ influx in Article Title: KCa3.1 Mediates Dysregulation of Mitochondrial Quality Control in Diabetic Kidney Disease. Article Snippet: Hence, KCa3.1 has been proposed as a potential therapeutic target for sickle cell anemia, autoimmunity, and Article Title: Functional Potassium Channels in Macrophages. Article Snippet: Macrophages are the predominant component of innate immunity, which is an important protective barrier of our body.. Macrophages are present in all organs and tissues of the body, their main functions include immune surveillance, bacterial killing, tissue remodeling and repair, and clearance of cell debris.. In addition, macrophages can present antigens to T cells and facilitate inflammatory response by releasing cytokines. Article Title: Targeting Effector Memory T Cells with a Selective Peptide Inhibitor of Kv1.3 Channels for Therapy of Autoimmune Diseases Article Snippet: It is noteworthy that it was 1600-fold Article Title: Protein Kinase A-Mediated Suppression of the Slow Afterhyperpolarizing KCa3.1 Current in Temporal Lobe Epilepsy Article Snippet: The KCa3.1 channel is ubiquitously expressed in the plasma membrane of neural and Article Title: The Kv1.3 K + channel in the immune system and its "precision pharmacology" using peptide toxins. Article Snippet: The complex interplay of ion channel activities generates a characteristic Ca2+ signal in activated T-cells (right middle of the cell) that regulates a plethora of cellular processes, leading to cell activation and cell type-specific effector functions 1 3 in activated CCR7− effector memory T (TEM) cells, whereas CCR7+ naïve and central memory T (TCM) cells preferentially express the Membrane:Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis. Article Snippet: Accepted Manuscript Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Govindaraj Anumanthan, Philip J. Wilson, Ratnakar Tripathi, Nathan P. Hesemann, Rajiv R. Mohan PII: S0014-4835(17)30534-1 DOI: 10.1016/j.exer.2017.12.003 Reference: YEXER 7243 To appear in: Experimental Eye Research Received Date: 26 July 2017 Accepted Date: 10 December 2017 Please cite this article as: Anumanthan, G., Wilson, P.J., Tripathi, R., Hesemann, N.P., Mohan, R.R., Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis, Experimental Eye Research (2018), doi: 10.1016/j.exer.2017.12.003.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Article Title: Blockade of KCa3.1: A novel target to treat TGF-β1 induced conjunctival fibrosis Article Snippet: KCa3.1 channel is the major contributor to Ca 2+ influx in Article Title: KCa3.1 Mediates Dysregulation of Mitochondrial Quality Control in Diabetic Kidney Disease. Article Snippet: Hence, KCa3.1 has been proposed as a potential therapeutic target for sickle cell anemia, autoimmunity, and Article Title: Functional Potassium Channels in Macrophages. Article Snippet: Macrophages are the predominant component of innate immunity, which is an important protective barrier of our body.. Macrophages are present in all organs and tissues of the body, their main functions include immune surveillance, bacterial killing, tissue remodeling and repair, and clearance of cell debris.. In addition, macrophages can present antigens to T cells and facilitate inflammatory response by releasing cytokines. Article Title: Targeting Effector Memory T Cells with a Selective Peptide Inhibitor of Kv1.3 Channels for Therapy of Autoimmune Diseases Article Snippet: It is noteworthy that it was 1600-fold Article Title: Protein Kinase A-Mediated Suppression of the Slow Afterhyperpolarizing KCa3.1 Current in Temporal Lobe Epilepsy Article Snippet: The KCa3.1 channel is ubiquitously expressed in the plasma membrane of neural and Article Title: The Kv1.3 K + channel in the immune system and its "precision pharmacology" using peptide toxins. Article Snippet: The complex interplay of ion channel activities generates a characteristic Ca2+ signal in activated T-cells (right middle of the cell) that regulates a plethora of cellular processes, leading to cell activation and cell type-specific effector functions 1 3 in activated CCR7− effector memory T (TEM) cells, whereas CCR7+ naïve and central memory T (TCM) cells preferentially express the |